Haplo-Insufficiency of Bcl2-Associated Athanogene 3 in Mice Results in Progressive Left Ventricular Dysfunction, β-Adrenergic Insensitivity, and Increased Apoptosis

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Author
McClung, Joseph M.
URI
http://hdl.handle.net/10342/9966
Subject
 BAG3, cardiomyopathy (DCM); genetic mutation; haplo-insufficiency 
Date
2018-03-30
Citation:
APA:
McClung, Joseph M.. (March 2018). Haplo-Insufficiency of Bcl2-Associated Athanogene 3 in Mice Results in Progressive Left Ventricular Dysfunction, β-Adrenergic Insensitivity, and Increased Apoptosis. Journal of Cellular Physiology, (233:9), p.6319-6326. Retrieved from http://hdl.handle.net/10342/9966

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MLA:
McClung, Joseph M.. "Haplo-Insufficiency of Bcl2-Associated Athanogene 3 in Mice Results in Progressive Left Ventricular Dysfunction, β-Adrenergic Insensitivity, and Increased Apoptosis". Journal of Cellular Physiology. 233:9. (6319-6326.), March 2018. December 06, 2022. http://hdl.handle.net/10342/9966.
Chicago:
McClung, Joseph M., "Haplo-Insufficiency of Bcl2-Associated Athanogene 3 in Mice Results in Progressive Left Ventricular Dysfunction, β-Adrenergic Insensitivity, and Increased Apoptosis," Journal of Cellular Physiology 233, no. 9 (March 2018), http://hdl.handle.net/10342/9966 (accessed December 06, 2022).
AMA:
McClung, Joseph M.. Haplo-Insufficiency of Bcl2-Associated Athanogene 3 in Mice Results in Progressive Left Ventricular Dysfunction, β-Adrenergic Insensitivity, and Increased Apoptosis. Journal of Cellular Physiology. March 2018; 233(9) 6319-6326. http://hdl.handle.net/10342/9966. Accessed December 06, 2022.
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