Involvement of RAB10 in Cognitive Dysfunction Using Trace Eyeblink Classical Conditioning

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dc.access.optionRestricted Campus Access Only
dc.contributor.advisorTran, Tuan
dc.contributor.authorHarris, Elizabeth
dc.contributor.departmentNeuroscience
dc.date.accessioned2023-07-26T16:53:43Z
dc.date.available2023-11-01T08:02:01Z
dc.date.created2023-05
dc.date.issued2023-05-04
dc.date.submittedMay 2023
dc.date.updated2023-06-30T13:44:47Z
dc.degree.departmentNeuroscience
dc.degree.disciplineMultidisciplinary Studies
dc.degree.grantorEast Carolina University
dc.degree.levelUndergraduate
dc.degree.nameBS
dc.description.abstractAlzheimer’s Disease (AD) is a disorder characterized by progressive memory loss, perceptive inaccuracies, and other issues with neurocognitive function (Khan et al., 2020). Different genetic issues and pathways in the brain may have a connection with AD. These include: Mutations in presenilin genes, hyperphosphorylated tau proteins, mutations in β-amyloid precursor protein and the accumulations of amyloid beta (Aβ) (Khan et al., 2020; Holtzman et al., 2011). Exploring these deficits may answer further questions surrounding AD.
dc.embargo.lift2023-11-01
dc.format.mimetypeapplication/pdf
dc.identifier.urihttp://hdl.handle.net/10342/13074
dc.publisherEast Carolina University
dc.subjectAlzheimer's, dementia, Rab10
dc.titleInvolvement of RAB10 in Cognitive Dysfunction Using Trace Eyeblink Classical Conditioning
dc.typeHonors Thesis
dc.type.materialtext

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